<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2176-6223</journal-id>
<journal-title><![CDATA[Revista Pan-Amazônica de Saúde]]></journal-title>
<abbrev-journal-title><![CDATA[Rev Pan-Amaz Saude]]></abbrev-journal-title>
<issn>2176-6223</issn>
<publisher>
<publisher-name><![CDATA[Instituto Evandro Chagas. Secretaria de Vigilância em Saúde e Ambiente. Ministério da Saúde]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2176-62232019000100023</article-id>
<article-id pub-id-type="doi">10.5123/s2176-6223201900096</article-id>
<title-group>
<article-title xml:lang="pt"><![CDATA[Hepatite C na década de 1980: resgate de casos das antigas hepatites "não A e não B" de um serviço de hepatologia na Amazônia, Brasil]]></article-title>
<article-title xml:lang="en"><![CDATA[Hepatitis C in the 1980s: case review of former non-A and non-B hepatitis from a hepatology service in the Brazilian Amazon]]></article-title>
<article-title xml:lang="es"><![CDATA[Hepatitis C en la década de 1980: rescate de casos de las antiguas hepatitis "no A y no B" de un servicio de hepatología en la Amazonía, Brasil]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Barbosa]]></surname>
<given-names><![CDATA[Kemere Marques Vieira]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Moreira]]></surname>
<given-names><![CDATA[Luiz Vinicius Leão]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Oliveira]]></surname>
<given-names><![CDATA[Candida Maria Abrahão de]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Souza]]></surname>
<given-names><![CDATA[Alex Junior Souza de]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Nunes]]></surname>
<given-names><![CDATA[Heloisa Marceliano]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Soares]]></surname>
<given-names><![CDATA[Manoel do Carmo Pereira]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Garcez]]></surname>
<given-names><![CDATA[Lourdes Maria]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Instituto Evandro Chagas/SVS/MS  ]]></institution>
<addr-line><![CDATA[Belém Pará]]></addr-line>
<country>Brasil</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidade do Estado do Pará  ]]></institution>
<addr-line><![CDATA[Belém Pará]]></addr-line>
<country>Brazil</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>00</month>
<year>2019</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>00</month>
<year>2019</year>
</pub-date>
<volume>10</volume>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://scielo.iec.gov.br/scielo.php?script=sci_arttext&amp;pid=S2176-62232019000100023&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.iec.gov.br/scielo.php?script=sci_abstract&amp;pid=S2176-62232019000100023&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://scielo.iec.gov.br/scielo.php?script=sci_pdf&amp;pid=S2176-62232019000100023&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="pt"><p><![CDATA[RESUMO  OBJETIVO: Descrever a frequência de infecção pelo vírus da hepatite C (VHC) em amostras de soro de pacientes diagnosticados como hepatite "não-A e não-B", atendidos em um instituto de pesquisa da Amazônia, entre 1982 e 1988.  MATERIAIS E MÉTODOS: Pesquisa descritiva, transversal e retrospectiva, incluindo 396 amostras de soro, preservadas a -20 °C e testadas para anticorpos IgG anti-VHC por ELISA. As amostras eram de pacientes de ambos os sexos, variando de 1 mês a 85 anos de idade, oriundos dos municípios de Belém e Ananindeua, Pará, Brasil. Para amostras anti-VHC reagentes, investigou-se a presença de RNA viral e, nos detectados, procedeu-se sequenciamento e genotipagem.  RESULTADOS: Anticorpos anti-VHC foram detectados em 10,9% (43/396) dos soros examinados. O RNA do VHC foi detectado em 55,8% (24/43) dos soros reagentes. Para 33,3% (8/24) dessas amostras, foi possível a distinção dos genótipos 1 (75,0%; 6/8) e 3 (25,0%; 2/8), com a confirmação de dois subtipos (1b e 3a).  CONCLUSÃO: Apesar das limitações, pelo longo tempo de armazenamento das amostras (três décadas), foi possível detectar e caracterizar o VHC em soros oriundos de biobanco, revelando a circulação de dois genótipos e dois subtipos virais à época: o genótipo 1, detectado na maioria das amostras genotipadas e, na atualidade, exibindo maior resistência a fármacos que os demais genótipos conhecidos; e o genótipo 3, menos frequente na amostra do estudo, associado a maior virulência. A análise permitiu identificar linhagens do VHC, favorecendo estudos futuros para elucidar aspectos evolutivos associados à resistência e à virulência desse patógeno.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[ABSTRACT  OBJECTIVE: To describe the frequency of Hepatitis C virus (HCV) infection in serum samples from patients diagnosed with "non-A and non-B" hepatitis attended at an Amazonian research institute between 1982 and 1988.  MATERIALS AND METHODS: Descriptive, cross-sectional, and retrospective research including 396 serum samples preserved at -20 °C and tested for anti-HCV IgG antibodies by ELISA. The samples were from patients of both sexes, ranging from 1 month to 85 years old, from Belém and Ananindeua, Pará State, Brazil. For reactive anti-HCV samples, the presence of viral RNA was investigated, and sequencing and genotyping were conducted in those detected.  RESULTS: Anti-HCV antibodies were detected in 10.9% (43/396) of the sera examined. HCV RNA was detected in 55.8% (24/43) of the reagent sera. For 33.3% (8/24) of these samples, it was possible to distinguish genotypes 1 (75.0%; 6/8) and 3 (25.0%; 2/8), with the confirmation of two subtypes (1b and 3a).  CONCLUSION: Despite the limitations, due to the long storage time of the samples (three decades), it was possible to detect and characterize HCV in biobank sera, revealing the circulation of two genotypes and two viral subtypes at the time: genotype 1, detected in most genotyped samples and currently exhibiting greater drug resistance than other known genotypes; and genotype 3, less frequent in the study sample, associated with higher virulence. The analysis allowed the identification of HCV strains, favoring future studies to elucidate evolutionary aspects associated with the resistance and virulence of this pathogen.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[RESUMEN  OBJETIVO: La infección frecuente por el virus de la hepatitis C (VHC) en muestras séricas de pacientes diagnosticados como hepatitis "no A y no B", observada en un instituto de investigación amazónico entre 1982 y 1988.  MATERIALES Y MÉTODOS: Investigación descriptiva, transversal y retrospectiva, incluyendo 396 muestras séricas, conservadas a -20 °C y probadas para anticuerpos Anti-VHC IgG por ELISA. Las muestras procedían de pacientes de ambos sexos, que oscilaban entre los 1 mes y los 85 años de edad, procedentes de los municipios de Belém y Ananindeua, Pará, Brasil. En el caso de las muestras reactivas contra el VHC, se investigó la presencia de ARN viral y, en la detección, se llevaron a cabo secuenciaciones y genotipado.  RESULTADOS: Se detectaron anticuerpos anti-VHC en el 10,9% (43/396) del suero examinado. El ARN del VHC se detectó en el 55,8% (24/43) de los sueros reactivos. Para el 33,3% (8/24) de estas muestras, fue posible distinguir los genotipos 1 (75,0%; 6/8) y 3 (25,0%; 2/8), con la confirmación de dos subtipos (1b y 3a).  CONCLUSIÓN: A pesar de las limitaciones, debido al largo tiempo de almacenamiento de las muestras (tres décadas), fue posible detectar y caracterizar el VHC en el suero del biobanco, revelando la circulación de dos genotipos y dos subtipos virales en ese momento: genotipo 1, detectado en la mayoría de las muestras genotipos y, hoy en día, exhibiendo una mayor resistencia a los fármacos que otros genotipos conocidos; y genotipo 3, menos frecuente en la muestra de estudio, asociado con una mayor virulencia. El análisis permitió la identificación de cepas de VHC, favoreciendo futuros estudios para dilucidar aspectos evolutivos asociados con la resistencia y virulencia de este patógeno.]]></p></abstract>
<kwd-group>
<kwd lng="pt"><![CDATA[Vírus da Hepatite C]]></kwd>
<kwd lng="pt"><![CDATA[Diagnóstico Molecular]]></kwd>
<kwd lng="pt"><![CDATA[Perfil Genotípico]]></kwd>
<kwd lng="pt"><![CDATA[Epidemiologia]]></kwd>
<kwd lng="en"><![CDATA[Hepatitis C Virus]]></kwd>
<kwd lng="en"><![CDATA[Molecular Diagnosis]]></kwd>
<kwd lng="en"><![CDATA[Genotypic Profile]]></kwd>
<kwd lng="en"><![CDATA[Epidemiology]]></kwd>
<kwd lng="es"><![CDATA[Virus de la Hepatitis C]]></kwd>
<kwd lng="es"><![CDATA[Diagnóstico Molecular]]></kwd>
<kwd lng="es"><![CDATA[Perfil genotípico]]></kwd>
<kwd lng="es"><![CDATA[Epidemiología]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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